CYBERMED LIFE - ORGANIC  & NATURAL LIVING

Convulsive Seizures

  • Effect of Sahaja yoga practice on seizure control&EEG changes in patients of epilepsy.

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    Abstract Title:

    Effect of Sahaja yoga practice on seizure control&EEG changes in patients of epilepsy.

    Abstract Source:

    Indian J Med Res. 1996 Mar;103:165-72. PMID: 9062044

    Abstract Author(s):

    U Panjwani, W Selvamurthy, S H Singh, H L Gupta, L Thakur, U C Rai

    Abstract:

    The effect of Sahaja yoga meditation on seizure control and electroencephalographic alterations was assessed in 32 patients of idiopathic epilepsy. The subjects were randomly divided into 3 groups. Group I (n = 10) practised Sahaja yoga for 6 months, Group II (n = 10) practised exercises mimicking Sahaja yoga for 6 months and Group III (n = 12) served as the epileptic control group. Group I subjects reported a 62 per cent decrease in seizure frequency at 3 months and a further decrease of 86 per cent at 6 months of intervention. Power spectral analysis of EEG showed a shift in frequency from 0-8 Hz towards 8-20 Hz. The ratios of EEG powers in delta (D), theta (T), alpha (A) and beta (B) bands i.e., A/D, A/D + T, A/T and A + B/D + T were increased. Per cent D power decreased and per cent A increased. No significant changes in any of the parameters were found in Groups II and III, indicating that Sahaja yoga practice brings about seizure reduction and EEG changes. Sahaja yoga could prove to be beneficial in the management of patients of epilepsy.

  • The ketogenic diet: mechanism of anticonvulsant action.

    Abstract Title:

    The ketogenic diet: mechanism of anticonvulsant action.

    Abstract Source:

    Adv Neurol. 1980 ;27:635-42. PMID: 6990715

    Abstract Author(s):

    C D Withrow

    Abstract:

    Although the ketogenic diet has been used in the therapy for epilepsy for more than 50 years, there are few studies concerned with the effects of this diet on the central nervous system. Recent attempts to unravel the biochemical effects of the ketogenic diet on the brain seem to be a fruitful approach to understanding how the ketogenic diet causes anticonvulsant effects. Another exciting approach is the development of animal models in which various effects of the diet can be correlated with changes in seizure protection. It would be useful to determine whether the diet produces any neurophysiological effects that could account for some, or all, of its antiseizure properties. Finally, the efficacy of the diet is impressive. It is likely that the ketogenic diet will never be of major therapeutic importance because of the expense and commitment required of the patient and the family. Nevertheless, it appears obvious that continued study of a therapy which seems to work so well will give us some valuable clues as to the mechanism of seizures and their control. Further investigations into the mechanism of action of the ketogenic diet should be encouraged.

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